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Institution	Date	Title	Author
國立臺灣大學	2004-12	Reactive oxygen species are crucial for hydroxychavicol toxicity toward KB epithelial cells	Jeng, J.H.; Wang, Y.L.; Chang, W.H.; Wu, H.L.; Li, C.H.; Uang, B.J.; Kang, J.J.; Lee, J.J.; Hahn, L.J.; Lin, B.R.; Chang, M.C.
國立臺灣大學	2009	Reactive Oxygen Species Are Involved in Fasl-Induced Caspase-Independent Cell Death and Inflammatory Responses	陳摘宇; 錢宗良; 林琬琬; CHEN, TSAI-YU; CHIEN, CHUNG-LIANG; LIN, WAN-WAN
國立臺灣大學	2009-03	Reactive oxygen species are involved in FasL-induced caspase-independent cell death and inflammatory responses	Chen, TY; Chi, KH; Wang, JS; Chien, CL; Lin, WW.
臺大學術典藏	2020-02-27T01:50:12Z	Reactive oxygen species are involved in FasL-induced caspase-independent cell death and inflammatory responses	Chen T.-Y.;Chi K.-H.;Wang J.-S.;Chung-Liang Chien;Lin W.-W.; Chen T.-Y.; Chi K.-H.; Wang J.-S.; CHUNG-LIANG CHIEN; Lin W.-W.
臺大學術典藏	2021-03-03T08:35:20Z	Reactive oxygen species are involved in FasL-induced caspase-independent cell death and inflammatory responses	Chen T.-Y.; Chi K.-H.; Wang J.-S.; Chien C.-L.; WAN-WAN LIN
國立臺灣大學	2008-12	Reactive oxygen species are involved in FasL-induced caspase-independent cell death and inflammatory responses.	Chen, TY; Chi, KH; Wang, JS; Chien, CL; Lin, WW
國立成功大學	2010/8/23	Reactive oxygen species are involved in regulating α1-adrenoceptor-activated vascular smooth muscle contraction	Tsai, Ming-Ho; Jiang, Meei Jyh
國立成功大學	2010/8/19	Reactive oxygen species are involved in regulating α1-adrenoceptor-activated vascular smooth muscle contraction	Tsai, Ming-Ho; Jiang, Meei Jyh
臺大學術典藏	2020-01-06T03:07:58Z	Reactive oxygen species are involved in shear stress-induced intercellular adhesion molecule-1 expression in endothelial cells	Chiu, J.J.; Wung, B.S.; Shyy, J.Y.J.; Hsieh, H.J.; Wang, D.L.; HSYUE-JEN HSIEH
臺大學術典藏	2020-01-06T03:08:01Z	Reactive oxygen species are involved in shear stress-induced intracellular adhesion molecule-1 (ICAM-1) expression in endothelial cells	Chiu, J. J.; Wung, B. S.; Hsieh, H. J.; Wang, D. L.; HSYUE-JEN HSIEH

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